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Nevertheless, it has also been demonstrated that glucose deficiency, activation of the system Xc system, high expression of SLC7A11, elevated NADP+/NADPH ratio, extensive NADPH consumption, cystine accumulation, and decreased GSH levels collectively drive ferroptosis [97]

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The contribution of this position to catalysis and binding is unknown, but given its proximity to both SA and the modeled glucose moiety of UDP-glucose, it could be crucial for orientation of the SA