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While the principles regulating glucose-dependence in cancer cells have been extensively reviewed, we still do not fully understand how cancer cells use many of the metabolic strategies to contribute to core metabolic functions in the presence of nutrients different from glucose

Jaspers, S., Hopermann, H., Sauermann, G., Hoppe, U., Lunderstdt, R., & Ennen, J

Pro-inflammatory cytokines like interleukin (IL)-6, IL-1 and tumor necrosis factor-alpha (TNF-) can cause degradation of tight junction proteins and the endothelial glycocalyx through activated MMPs and trigger a reactive phenotype of microglia [167,168,169] (Fig

The sensitivity of the forward rate constants for the superoxide formation reaction, k p2 , and the oxygen concentration parameter, k O2 , proved to be the most sensitive globally with respect to total effects, highlighted in Fig