Transcriptional profiling and biomarker identification reveal tissue specific effects of expanded ataxin-3 in a spinocerebellar ataxia type 3 mouse model
However, the single intervention was less promising when the host metabolic statuses were worsening with the likelihood of multiple nutrient insufficiencies, as in patients with an established diagnosis of metabolic or immune-related disorders
Oxidative damage and schizophrenia: an overview of the evidence and its therapeutic implications
ICH aetiology must be determined to assess the risk of recurrent ICH and other vascular events, which informs the use of individualized secondary prevention strategies and decisions on the use of antiplatelet or anticoagulant treatments for other indications
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