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The scientific literature tends to describe it as a modulatory peptide that nudges homeostatic processes rather than forcing sedation the way a benzodiazepine or a Z-drug hypnotic does

An in vitro model composed of senescent Leydig cells was used

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not established in humans

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Drosophila and mice with the Y314S variant both showed degeneration of dopaminergic neurons and locomotor defects (Lin et al., 2020), providing direct evidence for the involvement of cIII in PD pathogenesis