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Ferroptotic pores induce Ca(2+) fluxes and ESCRT-III activation to modulate cell death kinetics

Think of them as targeted messengers that tell your body what to fix and how to recover

Here's the mechanism: the enzyme ALDH2 (aldehyde dehydrogenase 2) oxidises acetaldehyde into acetic acid, but this process requires adequate glutathione reserves to neutralise reactive oxygen species (ROS) generated during the reaction

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A retrospective study on outcomes following posterior fossa decompression with dural splitting surgery in patients with Chiari type I malformation

Chronic inflammation caused by autoimmune diseases can increase oxidative stress