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Abstract Metabolomics studies in sickle cell disease (SCD) have been so far limited to tens of samples, owing to technical and experimental limitations

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Alternatively, in the absence of ACMSD activity, ACMS can spontaneously decay into quinolinic acid, a biosynthetic precursor to NAD + (see Fig

These findings were further highlighted by the identification of metabolic correlates to HbC % (Figure 2D), the top of which presented in the form of violin plots in Figure 2E