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Hence, we examined the effects of TAT-DAT NT in an acute dopamine depletion model

Mito-Met 10 ), with enhanced mitochondrial targeting capacity, have been demonstrated to enhance metformin-mediated inhibition of mitochondrial respiration and radiosensitisation in prostate cancer (57), suggesting that metformin-mediated modulation of mitochondrial metabolism is a key mechanism underlying its radiosensitising effects

These deposits consist of islet amyloidpolypeptide or amylin, that is believed, to originate in the -cell secretory granule

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Finally, metabolic disturbances, though inadequately reported, represent a theoretical risk