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In experimental systems, receptor activation by Cagrilintide has been associated with modulation of neuronal signaling pathways within hindbrain regions involved in metabolic regulation [1]

doi: 10.1159/000443344 3

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Additionally, upregulated Notch binding and metallopeptidase activities matched expression changes of TN-R (Notch-regulated ECM protein) and CTSS, potentially aggravating blood-brain barrier disruption via Notch-dependent microglial activation [67, 68] (Fig

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