Figure 2 Oxidative insult and male hormones in reproduction The occurrence of oxidative stress depends on either the overproduction of ROS or depletion of antioxidants, which may result in lipid peroxidation in Leydig cells and germ cells and is detrimental to lipoproteins, protein aggregation and fragmentation, and steroidogenic enzyme inhibition ( It has been noted that hormones, such as follicle stimulating hormone (FSH), luteinizing hormone (LH), testosterone, estrogen (E2) and prolactin (PRL), may regulate seminal total antioxidant capacity (TAC) ( As discussed above, excessive ROS influences the hypothalamic-pituitary adrenal axis (HPA) and in turn releases corticosterone and cortisol in animals and humans, which induces stress
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10.3177/jnsv.63.405 [DOI] [PubMed] [Google Scholar] Tan, T
PMID: 37108769 Int J Mol Sci Ferroptosis and Apoptosis Are Involved in the Formation of L-Selenomethionine-Induced Ocular Defects in Zebrafish Embryos
