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Nat Commun 9, 4130

This AnnAt1 regulatory mechanism could thus represent a way to mediate crosstalk between oxidative stress caused by ABA and Ca 2+ signaling pathways

Depleted glutathione by NAPQI leads to increased H 2 O 2 release that oxidizes thioredoxin and induces the disassociation of thioredoxin from apoptosis signalling-regulating kinase 1 (ASK-1), triggering self-activation of ASK-1, which phosphorylates mitogen-activated protein kinase kinase 4/7 and activates c-Jun N-terminal kinase (JNK) (Nakagawa et al., 2008)

Adeshirlarijaney, A., Zou, J., Tran, H

The iron found in hemoglobin is responsible for the red color of the blood

Woods, 2016