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The entry of the virus itself and the viral load can modulate oxidative stress and the inflammatory response, both processes that directly contribute to COVID-19 severity (Fig

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Furthermore, PGAM5 modulation of the anti-inflammatory response also inhibits the activation of proinflammatory-associated KCa3.1 potassium channels by dephosphorylating NDPK-B in CD4 + T cells 91

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