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demonstrated that oxidative stress and H 2 O 2 treatment led to a marked increase in senescence-associated -galactosidase activity but only to minimal apoptotic cell death in CSCs, suggesting that ROS triggered the induction of senescence and exhibited therapeutic potential in the eradication of drug-resistant CSCs [8]

Dixon, L

705 Albiflorin not only inhibits neuronal ferroptosis by reducing the levels of lipid peroxide and iron and regulating ferroptosis-related proteins, but also inhibits microglia activation and reduces the production of pro-inflammatory cytokines, thereby promoting motor function recovery after SCI in rats

doi: 10.1073/pnas.96.5.2356

This research was financially supported by Ningxia Natural Science Foundation (grant no

2022), while Quercetin and Myricetin interact with CYP1B1 and AKR1B1 to mitigate oxidative stress damage (Wang et al