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Oxidative stress was induced by tBuOOH (0.1, 0.3, 0.5, 0.7, 1, and 1.5 mM

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Reducing tissue damage and improving outcomes in ARDS may be achieved by inhibiting effector T cell ferroptosis

Mitochondrial impairment but not peripheral inflammation predicts greater Gulf War illness severity

Importantly, targeting cholesterol biosynthetic enzymes such as CYP51A1 can influence ferroptosis independently of metabolite levels, implying that these enzymes may have non-canonical, cholesterol-independent functions in ferroptotic regulation