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96 Meanwhile, high glucose and inflammation block LKB1 recruitment, activate PP2A/C, and induce degradation of AMPK catalytic subunits through the E3 ubiquitin ligase

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These reactive molecules can also form DNA adducts, which under repair impairment can lead to mutations and other genetic abnormalities

Importantly, we could not detect relevant amounts of CCR2+ cells (C-C chemokine receptor type 2, a typical marker of infiltrating monocytes and some neutrophil subsets [26]) although having established solid positive controls (Supplementary Fig