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FYCO1 regulates cardiomyocyte autophagy and prevents heart failure due to pressure overload in vivo

A major source of ROS during reperfusion is from the mitochondria, as a result of mitochondrial dysfunction

Puusepp, S

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However, CD3 CAR T cells of LTR exhibited significantly increased mTOR and pS6 expression compared to STR, suggesting a shift from a memory-to-effector metabolic phenotype of LTR CAR T cells from pre- to post-infusion, respectively (Fig