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This condition, often occurring during stroke or cardiac arrest, results from the restoration of blood flow to previously ischemic brain tissue, triggering a cascade of oxidative stress, inflammation, and neuronal death

Hereditary alpha tryptasemia: genotyping and associated clinical features

instead, research points to interactions with the nitric oxide (NO) system, growth factor receptor upregulation, and modulation of several cytokine pathways

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Leucine, a key amino acid from a signaling point of view, is an allosteric activator of GDH, inducing the production of -ketoglutarate and preventing GLS inhibition by glutamate accumulation [38]