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The hallmark pathological features of AD include the accumulation of -amyloid (A) peptides forming senile plaques and hyperphosphorylated tau aggregating into neurofibrillary tangles (NFTs), accompanied by synaptic dysfunction, neuronal loss, and chronic neuroinflammation (Agostinho et al., 2010

Ginsenoside metabolite compound K exerts joint-protective effect by interfering with synoviocyte function mediated by TNF-alpha and tumor necrosis factor receptor type 2

A: Many patients feel a boost in energy within hours, while mood and metabolism benefits may develop over several days