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doi: 10.1007/s11252-016-0555-z 57

In Figure 3B, the high fluorescence intensity of FITC-sOtx2-GHK and FITC-RGD-GHK were observed by CLSM, while FITC-GHK exhibited only modest fluorescence, and peptides colocalized with the cell membrane

Why are these injuries so notoriously stubborn

This imbalance between oxidation and reduction triggers autoimmune reactions that damage tissue structures

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In conclusion, advancing our understanding of circadian regulation of BBB repair will not only deepen insight into stroke pathogenesis but also enable the design of temporally targeted treatments