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As a result, hepatocellular supplies of glutathione become exhausted and NAPQI is free to react with cellular membrane molecules, resulting in widespread hepatocyte damage and death, leading to acute hepatic necrosis

cited 2025 Apr 21]

Lublin FD Coetzee T Cohen JA Marrie RA Thompson AJ International International Advisory Committee on Clinical Trials in MS

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The activity and expression of NRF2 were decreased in many AKI and CKD animal models, suggesting that NRF2 was an important mediator in the pathogenesis and progression of AKI and CKD [204, 205]