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Notably, perturbation of mitochondrial dynamics modulates this vulnerability: deletion of the fission regulator dynamin-related protein 1 (Drp1) prolongs m maintenance and confers resistance to ferroptotic death [103], whereas ferroptotic signaling is associated with Drp1 activation and mitochondrial fragmentation

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The pathways collectively contribute to metabolic regulation, anti-inflammatory effects, and tissue remodeling, thereby enhancing ovarian restoration

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