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Compared to mice with normal Gls1 expression, Gls1-deficient mice exhibited a significant increase in overall survival after radiotherapy, and their levels of natural killer cells and interferon alpha/gamma proteins were significantly higher than those of mice with normal Gls1 expression

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This refinement, which resulted in nine significant clusters, underscored the importance of balancing computational efficiency with result reliability, ensuring that the most promising inhibitors were prioritized for further analysis