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SARS-CoV-2 induces oxidative stress and ROS generation which can lead to several types of regulated cell death including NETosis, ferro

Selenoproteins such as GPXs and TRs can scavenge H2O2, protect cell membrane structure and function, repair the site of molecular damage, achieve anti-oxidative stress and local protective effects against oxidative stress or inflammation

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[4] The angiogenesis concern deserves specific attention because it is the most biologically plausible theoretical risk

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