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Reversibility and persistence of di-2-ethylhexyl phthalate (dehp)- and phenobarbitalinduced hepatocellular changes in rodents

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In individuals with obesity or metabolic dysfunction, amylin signaling may be impaired

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Interventions aimed at mitigating iron toxicity (e.g., chelation), selectively clearing pathological senescent cells or modulating their SASP (senotherapeutics), or directly inhibiting fibrotic pathways hold potential for developing more effective, non-hormonal treatments