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Evidence for novel biotransformation pathways involving quinone methide formation and thiazolidinedione ring scission

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Through the exosomeglialinflammation cascade, this mechanism facilitates the spread of focal neuronal injury to adjacent brain regions, thereby accelerating region-specific neurodegeneration and functional impairment in Alzheimers disease (Figure 3)

Allergic Reaction While uncommon, some patients are allergic to glutathione

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Increased post-mitotic senescence in aged human neurons is a pathological feature of Alzheimer's disease