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GSH, exploiting the redox-active thiol residue (-SH) of cysteine, exerts its antioxidant function mainly via Gpxs-mediated reactions, which result in peroxide buffering with simultaneous oxidation of GSH to GSSG (19)

Downregulated connexin-43 expression leads to vascular cell death and increases vascular permeability in the retina [114]

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Nrf2, in turn, can stimulate p62 gene expression thus amplifying a pro-survival circuit that can favour tumourigenesis