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doi: 10.1146/annurev-cellbio-100913-013325 154 ItohN.ItohY.TassoniA.RenE.KaitoM.OhnoA.et al
Interestingly, these events were shown to be independent of calpain activity, a calcium-dependent protease also implicated in cell death processes
Therefore, the authors suggested that the involvement of these GSTs in lambda -cyhalothrin resistance might be through sequestration [117]
In LKB1-deficient non-small cell lung cancer (NSCLC) models, AMPK inactivation derepresses CPS1, whose silencing induces nucleotide pool imbalance (reduced pyrimidine/purine ratio), S-phase arrest, and DNA damage, mechanisms hypothesized to operate in PCa, where ammonium metabolism inhibitors (e.g., targeting glutaminase) combined with autophagy modulators (e.g., ULK1 activators) could exploit metabolic vulnerabilities targeting tumor cells (200)
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