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Deficiency of sting promotes collagen-specific antibody production and B cell survival in collagen-induced arthritis

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This difference was also confirmed by another GSH specific reagent, namely thiol green dye (Supplementary Figure 4)

Therefore, alternative therapeutic targets other than NRF2 itself are required for controlling NRF2-addicted cancer cells

Namely, sustained activation of PI3K-AKT pathway dramatically promotes the accumulation of NRF2 by inhibiting KEAP1-independent degradation mechanism of NRF2, resulting in the augmentation of NRF2-mediated transcriptional activation (Mitsuishi et al