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Microglial cells, which function like macrophages in the CNS, play a crucial role in the brain's innate defense mechanisms and are primarily responsible for inflammation-mediated neurodegeneration

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OA-VI12 was shown to promote the expression of miR-122-5p while downregulating the expression of MITF and TYR, suggesting that it inhibited melanin synthesis through the miR-122-5p/MITF/TYR axis