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10.1002/smll.201001886 85 PrietoT.MarconR

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Both in vivo and in vitro, GSH treatment fully blocked TGF/SMAD pathway activation by inhibiting excessive ROS production

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158 The ability of eNAMPT to trigger this IL-6/STAT3 antiapoptotic loop was not dependent on the presence of nicotinamide (the NAMPT substrate), was not reproduced by nicotinamide mononucleotide (NMN), the product of NAMPT and was not blocked by the enzyme inhibitor FK866