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Elevated pro-inflammatory cytokines, tumor necrosis factor-alpha (TNF-), interleukin (IL)1, and IL-6 in patients with depression may disrupt emotional regulation by crossing the bloodbrain barrier [15], while depressive behaviors can further exacerbate inflammation [16]

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[4] [5] This activation has been observed in preclinical studies to influence glucose uptake, lipid metabolism, and mitochondrial function

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doi: 10.1016/j.ymgme.2011.04.010 145