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In addition, an increasing amount of research has supported a pathophysiological role for ferroptosis in the development of cardiovascular diseases, including myocardial ischemiareperfusion injury, myocardial infarction, adriamycin-mediated myocardial injury, and heart failure [69]

& Koudriavtseva, T

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A 50 mg dose equals 10 mL which is the entire vial reconstituted at 5 mg/mL

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However, the suitability of current in vitro models, including those based on ARPE-19 cells, for accurately recapitulating RPE physiology in the context of diabetes warrants further optimization