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It has been found that cancer-associated fibroblasts (CAFs) have a greater dependence on Gln than tumor cells do and exhibit greater sensitivity to GLS inhibition (51)

We found that two key metabolic mediators, acetyl-CoA acetyltransferase 2 (Acat2) and ATP-citrate lyase (Acly), previously shown to control T H 17 cell effector function through glycolytic-epigenetic reprogramming 21 , were downregulated in CD38- compared to IgG-treated 5xFAD mice, 1 month after initiation of the treatment (Fig

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