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Tan IIA corrects the abnormal autophagic flux induced by DOX through a dual mechanism involving enhanced autophagosome formation and autolysosomal degradation, thereby improving cardiomyocyte viability (53)

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Nonketotic hyperosmolar coma and frusemide therapy

During sepsis, both the synthesis and bioavailability of GSH are significantly impaired, leading to uncontrolled ROS accumulation and cellular injury (Table 2)

This photothermally-enhanced approach effectively facilitates the repair of mitochondrial damage and downregulates the NF-B signaling pathway to inhibit the expression of pro-inflammatory cytokines

Vildagliptin attenuates Huntingtons disease through activation of GLP-1 receptor/PI3K/Akt/BDNF pathway in 3-nitropropionic acid rat model