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The ototoxicity of cisplatin is permanent sensorineural hearing loss due to the major damage to the spiral apparatus (Corti organ), spiral ganglion neurons (SGN) and vascular striae (Rybak et al., 2019

Decreases were also detected in 1- and 4-month-olds at 3 hours after CCI injury (Durham et al., 2000)

This study revealed that chlorine sensing signal pathways promte an anti-inflammatory responses by microglia, reducing injury and inflammation after stroke

However, investigations conducted to date have been through animal or in vitro studies, which have produced largely conflicting results

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Due et al., Evidence supports that after a central nervous system injury, increased extracellular glutamate levels activate several intracellular pathways including ROS formation