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Binding to the IGF1R initiates intracellular signaling

Mechanistically, the upregulation of OCTN2 promotes the proliferation and migration of HCC cells in vitro and augments the growth and metastasis of HCC, as well as the cancer stem-like properties of HCC by increasing FAO and OXPHOS, which depends on PGC-1 signaling

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Additionally, the therapeutic impact of APS on NAFLD may be related to its capacity to ameliorate OS, ERS, and mitochondrial damage through regulation of the NRF2/HO-1, GRP78/IRE-1/JNK, and AMPK/PGC-1/NRF1 pathways, as seen in Figure 3

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The COMT gene catechol-O-methyltransferase helps to break down and expel substances that have a catechol structure