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The cells were collected, washed and ROS was quantified on a NovoCyte flow cytometer, with an excitation max of 640 nm and emission max of 665 nm

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These data suggest that both the inhibition of mTOR pathway and the activation of FoxO1 pathway both depend on ECM1-LRP1 interaction, ultimately preventing HSC activation and reducing liver fibrosis in mice

Spiller HA (2017) Rethinking mercury: the role of selenium in the pathophysiology of mercury toxicity