Metabolic enzymes essential for viral replication are enriched among the interactome of human-pathogenic viruses In the next step, we used our models to identify potential metabolic targets to inhibit viral replication
Ilie MD, Villa C, Cuny T, Cortet C, Assie G, Baussart B, et al
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34: 8997 Bernardini I, Rizzo WB, Dalakas M, et al
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This pairing allows researchers to study whether dual-pathway targeting (amylin + GLP-1) produces superior outcomes compared to single-agent protocols, with cagrilintide addressing gastric motility while semaglutide modulates incretin signalling independently