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Thus, copper toxicity may be a process that is dependent on GSH depletion, and indeed, increased concentrations of copper and dysregulation of copper homeostasis are observed in several neurodegenerative diseases in which GSH status is reduced, including Alzheimer's disease and models of ALS [54, 56]

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More recently, a meta-analysis study linked GSTT1-1 null polymorphism with an increased risk of coronary heart disease 43

Thirty unfed nymphs were used for each concentration and divided into 3 replicates (10/replicate)

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