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It is likely that the particular molecular pathways[11,25-29] that have been suggested to be involved in the healing effects of BPC 157, as well as its recently demonstrated ability to cause the increased expression and internalization of VEGFR2 and the activation of the VEGFR2-Akt-eNOS signaling pathway[25], contribute to its action

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It is commonly studied where peptide signaling, cytoskeletal activity, and cellular migration are evaluated together under conditions of mechanical stress or localized disruption