(NCM)] were added to microglial cultures following H/I, BDNF released from microglia was upregulated, suggesting that BDNF might contribute to the anti-inflammatory activity induced by microglia (61)
The pNRF2 channel was held to a consistent threshold within each replicate and the pNRF2 area fraction for each nucleus was recorded using the ROI manager
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This activation enhanced host antioxidant capacity and mitigated postpartum oxidative stress 86,87
This result would not survive multiple comparisons between different metabolites