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M.GarssenJ.Van BergenhenegouwenJ.OverbeekS

Molecular Cell Biology

It facilitate A clearance by upregulating neprilysin expression and activating autophagy, while also inhibiting tau hyperphosphorylation via modulation of kinases such as GSK3 (ElSayed and Bayan 2015

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1) are dysregulated in response to the GDH2 gene deletion, or these pathways are unable to generate sufficient glutamate inside the cells and the large pool of free NADP + required to overcome the thermodynamic barrier inherent in using the still present Gdh3, which is usually an anabolic enzyme with a higher affinity for -ketoglutarate than for glutamate

These combined actions underscore their therapeutic potential for NAFLD (89, 90)