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The downregulation of MT1G, caused by hypermethylation of its promoter, is associated with the maintenance of pancreatic cancer stemness

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The challenge is not whether GHK-Cu has biological activity (that is well-established) but whether that activity can be delivered to tissues at concentrations and durations sufficient to produce clinical effects beyond the skin surface

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doi: 10.1016/j.diabres.2022.110202