Evidence for oxidative pathways in the pathogenesis of PD: are antioxidants candidate drugs to ameliorate disease progression
Methylation includes in this box inFIGS
indicates that PAC, in the absence of adequate, physiological levels of GSH, may give rise to genotoxic quinone imine metabolites ( The production of quinone imine metabolite is the primary responsible for PAC liver and kidney toxicity
This results in the inactivation of NF-B-dependent inflammatory factors such as TNF- and IL-6, as well as the blockade of cell signaling molecules such as MAP kinases
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For instance, activated macrophages and T cells often exhibit heightened glycolysis and altered mitochondrial function, affecting the synthesis of NADPH and GSH (136, 137)
