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However, this review has limitations, notably its inability to comprehensively cover the specific molecular mechanisms of ferroptosis across different OA subtypes, and it provides limited discussion on the interactions between ferroptosis and other forms of cell death

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[3] The regulation of levels of ROS, levels of oxidized and reduced glutathione and other thiols, and antioxidant enzymes (such as GSTs and glutathione reductase) are important in determination of the most stable conditions for redox control, or for activation of apoptosis

Archived from the original on 26 May 2015

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