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An early publication in the Annals of the New York Academy of Sciences documenting the original discovery of telomere shortening as a primary mechanism of cellular senescence and its relevance to human aging biology provides the foundational framework for understanding why telomerase-activating compounds attract longevity research attention

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Colocalization analyses have pinpointed genetically correlated loci harboring putatively shared causal variants for both MDD and T2DM [57]

2015;5: a021196

Supplementary Material Acknowledgments We thank Dr Erkki Ruoslahti of the Sanford-Burnham Medical Research Institute for providing us with a peptide display phage library and valuable suggestions