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Firstly, we verified across a panel of normal and cancer cell lines with differing p53 status (Supplementary Table 1) that treatment with a lethal dose of APR-246 resulted in higher intracellular ROS (Fig

Journal of Autism and Developmental Disorders, 49 (5), 17781794

However, it does not stop: Hydrolysis Oxidation Deamidation Natural chemical degradation Chemical stability and microbial protection are not the same thing

Together, these results indicate that decreased GSH/GSSG redox/antioxidant capacity and increased oxidative stress in the autism brain may have functional consequence in terms of a chronic inflammatory response, increased mitochondrial superoxide production, and oxidative protein and DNA damage

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