The formation of the ATG16L1 complex is initiated by activation of the ubiquitin-like protein ATG12 by ATG7 (E1-like enzyme) in an ATP-dependent manner, resulting in the formation of a covalent bond between ATG12 and ATG7
Treatment risks and logistics do not diminish interest in antiamyloid medication
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Do not freeze it once it's in liquid form, as the freeze-thaw cycle can damage the peptide structure
Mercado, A., Broumand, V., Zandi-Nejad, K., Enck, A
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