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[7] Because it engages GIP and GLP-1 within one molecule, it is mechanistically distinct from CagriSema, which recruits amylin rather than GIP

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GCK also exerts joint protection by inhibiting the proliferation, migration and secretion of synovial cells (129), whose molecular mechanism is to inhibit the secretion of TNF- in AIA-FLS rats, down-regulate the expression of tumor necrosis factor receptor type 2 (TNFR2), and inhibit TNF--mediated proliferation, migration and secretion of AIA -FLS

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Thymosin Beta-4 cell migration and inflammation resolution