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The central mechanism under investigation is its ability to stimulate telomerase, an enzyme responsible for maintaining the length of telomeres

The second part of the study began by randomly dividing the test subjects in three groups including: APP/PS1, Dihexa administered at a dose of 2.88 mg/kg, and 2.88 mg/kg of Dihexa + 0.5 mg/kg of wortmannin

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More specifically, it causes the active forms to convert to cyanocobalamin, causing a functional deficiency