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corpus callosum in CAE), in the former, a deliberately orchestrated primary autoimmune process directed at myelin antigens results in lymphocytic infiltration, secondary injury to white matter and a universal innate response, whereas in CAE, where the primary insult is a biochemical one deliberately targeting intrinsic CNS myelin, following immune stimulation, the resulting lesion is pathologically similar (Day, 2005

At the 5mg/ml concentration, the same one-unit difference is only 0.05mg, a 2% error

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After six months, there were no significant changes in tinnitus severity in any of the treatment groups

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They allow adjustments to peptide concentrations, dosage, and formulations, making it easier to align the experiment design with specific research goals